Welcome to BB Eye Foundation VIP Kolkata


Glaucoma is a group of eye diseases which result in damage to the optic nerve and vision loss. The most common type is open-angle glaucoma and less common types including closed-angle glaucoma and normal-tension glaucoma. Open-angle glaucoma develops slowly over time and there is no pain. Side vision may begin to decrease followed by central vision resulting in blindness if not treated. Closed-angle glaucoma can present gradually or suddenly. The sudden presentation may involve severe eye pain, blurred vision, mid-dilated pupil, redness of the eye, and nausea. Vision loss from glaucoma, once it has occurred, is permanent.


Dietary: No evidence indicates vitamin deficiencies cause glaucoma in humans. It follows, then, that oral vitamin supplementation is not a recommended treatment for glaucoma. Caffeine increases intraocular pressure in those with glaucoma, but does not appear to affect normal individuals.

Ethnicity: Many people of East Asian descent are prone to developing angle closure glaucoma because of shallower anterior chamber depths.


pseudoexfoliation glaucoma the pressure is due to the accumulation of microscopic granular protein fibres, which can block normal drainage of the aqueous humour. PEX is prevalent in Scandinavia, primarily in those over 70, and more commonly in women.

Pigmentary glaucoma is caused by pigment cells sloughing off from the back of the iris and floating around in the aqueous humour. Over time, these pigment cells can accumulate in the anterior chamber in such a way that it can begin to clog the trabecular meshwork. It is a rare condition that occurs mostly among Caucasians, mostly males in their mid-20s to 40s, and most are nearsighted.Primary juvenile glaucoma is a neonate or juvenile abnormality where ocular hypertension is evident at birth or shortly thereafter and is caused by abnormalities in the anterior chamber angle development that blocks the outflow of the aqueous humour.

Uveitis Glaucoma is due to uveitis, the swelling and inflammation of the urea, the middle layer of the eye. The urea provides most of the blood supply to the retina. Increased eye pressure in uveitis can result from the inflammation itself or from the steroids used to treat it.


LASER: Argon laser trabeculoplasty (ALT) may be used to treat open-angle glaucoma. It is a temporary solution, not a cure. A 50-μm argon laser spot is aimed at the trabecular meshwork to stimulate the opening of the mesh to allow more outflow of aqueous fluid. Usually, half of the angle is treated at a time. Traditional laser trabeculoplasty uses a thermal argon laser in an argon laser trabeculoplasty procedure.

A newer type of laser trabeculoplasty uses a "cold" (nonthermal) laser to stimulate drainage in the trabecular meshwork. This newer procedure, selective laser trabeculoplasty (SLT), uses a 532-nm, frequency-doubled, Q-switched Nd:YAG laser, which selectively targets melanin pigment in the trabecular meshwork cells. Studies show SLT is as effective as ALT at lowering eye pressure. In addition, SLT may be repeated three to four times, whereas ALT can usually be repeated only once.

Nd:YAG laser peripheral iridotomy (LPI) may be used in patients susceptible to or affected by angle closure glaucoma or pigment dispersion syndrome. During laser iridotomy, laser energy is used to make a small, full-thickness opening in the iris to equalize the pressure between the front and back of the iris, thus correcting any abnormal bulging of the iris. In people with narrow angles, this can uncover the trabecular meshwork. In some cases of intermittent or short-term angle closure, this may lower the eye pressure. Laser iridotomy reduces the risk of developing an attack of acute angle closure. In most cases, it also reduces the risk of developing chronic angle closure or of adhesions of the iris to the trabecular meshwork. Diode laser cycloablation lowers IOP by reducing aqueous secretion by destroying secretory ciliary epithelium.

Please find below our doctor's details with information related to their sepcialization

Doctors Name Telephone Email Slot
DR. CHANDRIMA PAUL 9830079189 drchandrimapaul@gmail.com TUESDAY - 9 A.M. - 11 A.M.
DR. MANEESH SINGH 9874439193 docmsingh@gmail.com MONDAY - 9 A.M.- 4 P.M.
DR. NILAY ACHARYA 9830152599 nila7553@gmail.com TUESDAY - 3 P.M. - 5 P.M.
DR. RITUPARNA DEY 9830498571 - THURSDAY - 10 A.M. - 4 P.M.
DR. SAGAR BHARGAVA 9874176663 sagarbhargava@yahoo.com TUESDAY/ FRIDAY AND SATURDAY - 11 A.M.- 4 P.M.
DR. SOUVIK BANERJEE 9883083083 - THURSDAY - 10 A.M. - 12.30 P.M.